Labchart reader finding slope12/27/2022 Elsevier, Amsterdam, pp 403–430īrand MD (1990) The Contribution of the leak of protons across the mitochondrial inner membrane to standard metabolic rate. In: Hoar WS, Randall DJ (eds) Fish physiology. īoutilier RG, Heming TA, Iwama GK (1984) Appendix: physicochemical parameters for use in fish respiratory physiology. Monterey, CA, USAīohning DE, Carter B, Liu S, Pohost GM (1990) PC-Based system for retrospective cardiac and respiratory gating of NMR data. In: The ocean in a high-CO 2 world: third symposium. (02)00482-4īock C, Dogan F, Pörtner H-O (2012) Cardiovascular performance of the edible crap Cancer pagurus under the effects of ocean acidification. īock C, Sartoris F-J, Pörtner H-O (2002) In vivo MR spectroscopy and MR imaging on non-anaesthetized marine fish: techniques and first results. (01)00414-3īock C, Sartoris F-J, Wittig R-M, Pörtner H-O (2001b) Temperature-dependent pH regulation in stenothermal Antarctic and eurythermal temperate eelpout (Zoarcidae): an in-vivo NMR study. īock C, Frederich M, Wittig R-M, Pörtner H-O (2001a) Simultaneous observations of haemolymph flow and ventilation in marine spider crabs at different temperatures: a flow weighted MRI study. īelman BW (1975) Some aspects of the circulatory physiology of the spiny lobster Panulirus interruptus. Rapid acquisition with relaxation enhancement RMR:Īppelhans YS, Thomsen J, Pansch C et al (2012) Sour times: seawater acidification effects on growth, feeding behaviour and acid–base status of Asterias rubens and Carcinus maenas. − log 10 of dissociation constant in an acid–base equilibrium \(\) Partial pressure of CO 2 in haemolymph P(CO 2) w Solubility coefficient level of significance ADP: This previously unknown phenomenon should direct attention to pathways of acid–base regulation and their potential feedback on whole-animal energy demand, in relation with changing seawater carbonate parameters. Our findings suggest an influence of water bicarbonate levels on metabolic rates as well as on correlations between blood flow and pH e. Despite similar levels of haemolymph pH and ion concentrations under OA, metabolic rates, and haemolymph flow were significantly depressed by 40 and 30%, respectively, when OA was combined with reduced seawater and pH. While extracellular cation concentrations increased throughout, anion levels remained constant or decreased. This process was effective even under reduced seawater pH and bicarbonate concentrations. High water P(CO 2) caused haemolymph P(CO 2) to rise, but pH e and pH i remained constant due to increased haemolymph and cellular. Cardiovascular performance was determined together with extra-(pH e) and intracellular pH (pH i), oxygen consumption, haemolymph CO 2 parameters, and ion composition. For a detailed understanding of the dependence of acid–base regulation on water parameters, we investigated the physiological responses of the shore crab Carcinus maenas to 4 weeks of ocean acidification, at variable water bicarbonate levels, paralleled by changes in water pH. Successful maintenance of body fluid pH depends on the functional capacity of ion-exchange mechanisms and associated energy budget. Acid–base regulation in gill breathers involves a net increase of internal bicarbonate levels through transmembrane ion exchange with the surrounding water. For more information, visit the JACC: Basic to Translational Science author instructions page.Ocean acidification causes an accumulation of CO 2 in marine organisms and leads to shifts in acid–base parameters. The authors attest they are in compliance with human studies committees and animal welfare regulations of the authors’ institutions and Food and Drug Administration guidelines, including patient consent where appropriate. Gregory Lewis, MD, served as guest editor for this paper. Wagner is serving as an expert witness in a legal case of scientific misconduct. Poole is supported by NIH grant HL-2-108328. Kelly is supported by NIH grants R01 DK045416, R01 HL058493, and R01 HL128349 and has received advisory board honoraria from Pfizer (significant) and Amgen (modest). Margulies is supported by NIH grants U10-HL110338, R01HL121510, and R01HL133080 and receives research funding from Sanofi (significant), Merck (significant), and GlaxoSmithKline (significant). Mazurek has received advisory board honoraria from Actelion Pharmaceuticals (modest) and United Therapeutics (modest). Zamani is supported by NIH grant 5-K23-HL130551 and has consulted for Vyaire (modest). The project described was supported by the National Center for Research Resources and the National Center for Advancing Translational Sciences, National Institutes of Health (NIH), through grant UL1TR001878.
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